I want discover the effects benzos, ambien or traqueilerz (or other sedating drugs) have on the stritatal large as aspiny cholinergic interneurons (LAIs) and low-threshold spike interneurons (LTSIs). I also wanted to know how or If they have any effects on the hyperpolarization of MSNs. I also want to figure out if FSIs are excited or inhibited by these substances. I want to know the effects on the firing rate of MSNSs. I want to show the relationship between the psychomotor and sedation effects and even memory impairment effects of these drugs and the firing rate of specifi striatal cell population. Striatal FSIs may have an important role in the behavioral effects of these drugs, and thus may be a valuable target in the development of novel therapies. Here’s a proposal for a publication that I made in 5 mins while im high as fuck. Though I did do research before hand. Please let me know if my reserach paper makes sense and if its worth mentioning to my Prof (Phd for Harvard, hola). And don't make fun of me if this doesn't make any sense.

http://www.berkelab.org/BerkeLab/Research_%26_Publications.html Heres a link to the lab I work in if you're interested in the publications. Thanks! (Second post ever by the way)

Upvotes(uptokes haha man im on r/trees to much) are much appreciated, I'm anything but a karma whore, I just want as many people as possible to see this post so I can get a wide range of input.


THANKS!